cygb (Proteintech)
Structured Review

Cygb, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 12 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+cygb/Cytoglobin+Antibody/pm39503803-59-54-58
Average 93 stars, based on 12 article reviews
Images
1) Product Images from "Cytoglobin augments ferroptosis through autophagic degradation of ferritin in colorectal cancer cells."
Article Title: Cytoglobin augments ferroptosis through autophagic degradation of ferritin in colorectal cancer cells.
Journal: Molecular and cellular biochemistry
doi: 10.1007/s11010-024-05148-0
Figure Legend Snippet: Fig. 5 CYGB combined TFR to regulate ferroptosis. (A) Western blotting analysis of FLAG-CYGB-overexpression in HCT116 and SW620. (B) The immunoprecipitation and immunoblotting were per- formed using FLAG-CYGB and TFR antibodies in HCT116. (C) Co-localization of TFR and FLAG-CYGB in HCT116 cells based on immunofluorescence staining
Techniques Used: Western Blot, Over Expression, Immunoprecipitation, Immunofluorescence, Staining
Related Articles
Membrane:Article Title: Glycyrrhizic acid ameliorates hepatic fibrosis by inhibiting oxidative stress via AKR7A2. Article Snippet: Background: Hepatic fibrosis is a reversible pathological phenomenon caused by the abnormal proliferation of connective tissues in the liver for self-repair after persistent liver injury.. Among these tissues, the activation status of hepatic stellate cells (HSCs) is crucial.. Glycyrrhizic acid (GA) agents have been proven to have excellent anti-fibrosis effects, but their targets are unclear. Labeling:Article Title: Glycyrrhizic acid ameliorates hepatic fibrosis by inhibiting oxidative stress via AKR7A2. Article Snippet: Background: Hepatic fibrosis is a reversible pathological phenomenon caused by the abnormal proliferation of connective tissues in the liver for self-repair after persistent liver injury.. Among these tissues, the activation status of hepatic stellate cells (HSCs) is crucial.. Glycyrrhizic acid (GA) agents have been proven to have excellent anti-fibrosis effects, but their targets are unclear. Incubation:Article Title: Glycyrrhizic acid ameliorates hepatic fibrosis by inhibiting oxidative stress via AKR7A2. Article Snippet: Background: Hepatic fibrosis is a reversible pathological phenomenon caused by the abnormal proliferation of connective tissues in the liver for self-repair after persistent liver injury.. Among these tissues, the activation status of hepatic stellate cells (HSCs) is crucial.. Glycyrrhizic acid (GA) agents have been proven to have excellent anti-fibrosis effects, but their targets are unclear. |